Interesting.... we all know someone who has had stents and my thoughts would be like, aren’t they fortunate that stents are available 😁 after this explanation I’ve learned “that all is not what it seems to be”🫣 thanks for the continued education 👍👍
On a not-very-related note: Any chance of doing a piece on coronary collateral circulation? As in: "I have just found out from CTCA that I have 3-vessel heart disease but have no symnptoms,low hsCRP, pulled 12 METs on the exercise stress test etc etc I also have an occluded left SFA but have developed a great leg collateral circulation system which lets me get ~70 Met hours of PA per week. Maybe the same kind of thing has happened with my heart and if so what implications for risk/benefit CABG analysis?"
Obviously I'm talking with cardio about this but would be great to get an independent survey of the evdience base, such as it is.
The engineer in me is thinking there must be a dynamic component to this if structural modifications to arterial wall lumen are comparable to pharmacological interventions? Is it likely that there are a number of biochemical/hormonal etc moderators (identified or not?) influencing the physiology?
I'm sure cleverer people them me have thought that!
Interesting.... we all know someone who has had stents and my thoughts would be like, aren’t they fortunate that stents are available 😁 after this explanation I’ve learned “that all is not what it seems to be”🫣 thanks for the continued education 👍👍
Great!
On a not-very-related note: Any chance of doing a piece on coronary collateral circulation? As in: "I have just found out from CTCA that I have 3-vessel heart disease but have no symnptoms,low hsCRP, pulled 12 METs on the exercise stress test etc etc I also have an occluded left SFA but have developed a great leg collateral circulation system which lets me get ~70 Met hours of PA per week. Maybe the same kind of thing has happened with my heart and if so what implications for risk/benefit CABG analysis?"
Obviously I'm talking with cardio about this but would be great to get an independent survey of the evdience base, such as it is.
The engineer in me is thinking there must be a dynamic component to this if structural modifications to arterial wall lumen are comparable to pharmacological interventions? Is it likely that there are a number of biochemical/hormonal etc moderators (identified or not?) influencing the physiology?
I'm sure cleverer people them me have thought that!